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<OAI-PMH schemaLocation=http://www.openarchives.org/OAI/2.0/ http://www.openarchives.org/OAI/2.0/OAI-PMH.xsd> <responseDate>2018-01-15T18:28:32Z</responseDate> <request identifier=oai:HAL:hal-01174251v1 verb=GetRecord metadataPrefix=oai_dc>http://api.archives-ouvertes.fr/oai/hal/</request> <GetRecord> <record> <header> <identifier>oai:HAL:hal-01174251v1</identifier> <datestamp>2018-01-11</datestamp> <setSpec>type:ART</setSpec> <setSpec>subject:sde</setSpec> <setSpec>collection:UNIV-PERP</setSpec> <setSpec>collection:CNRS</setSpec> <setSpec>collection:SDE</setSpec> <setSpec>collection:EPHE</setSpec> <setSpec>collection:GIP-BE</setSpec> <setSpec>collection:CRIOBE</setSpec> <setSpec>collection:AGROPOLIS</setSpec> <setSpec>collection:UNIV-AG</setSpec> <setSpec>collection:UNIV-NC</setSpec> <setSpec>collection:EHESS</setSpec> <setSpec>collection:IFREMER</setSpec> <setSpec>collection:IRD</setSpec> <setSpec>collection:PSL</setSpec> <setSpec>collection:UNIV-POLYNESIE</setSpec> <setSpec>collection:UPF</setSpec> </header> <metadata><dc> <publisher>HAL CCSD</publisher> <title lang=en>Histopathology of crustose coralline algae affected by white band and white patch diseases</title> <creator>Quéré, Gaëlle</creator> <creator>Meistertzheim, Anne-Leila</creator> <creator>Steneck, Robert S., </creator> <creator>Nugues, Maggy M.</creator> <contributor>Leibniz Centre for Tropical Marine Research (ZMT)</contributor> <contributor>Laboratoire d'Excellence CORAIL (LabEX CORAIL) ; Institut de Recherche pour le Développement (IRD) - Université des Antilles et de la Guyane (UAG) - École des hautes études en sciences sociales (EHESS) - École pratique des hautes études (EPHE) - Institut Français de Recherche pour l'Exploitation de la Mer (IFREMER) - Université de la Réunion (UR) - Université de la Polynésie Française (UPF) - Université de Nouvelle Calédonie - Institut d'écologie et environnement</contributor> <contributor>Centre de recherches insulaires et observatoire de l'environnement (CRIOBE) ; Université de Perpignan Via Domitia (UPVD) - École pratique des hautes études (EPHE) - Centre National de la Recherche Scientifique (CNRS)</contributor> <contributor>Darling Marine Center ; University of Maine</contributor> <contributor>Caribbean Research and Management of Biodiversity Foundation (CARMABI)</contributor> <description>International audience</description> <source>ISSN: 2167-8359</source> <source>PeerJ</source> <publisher>PeerJ</publisher> <identifier>hal-01174251</identifier> <identifier>https://hal-univ-perp.archives-ouvertes.fr/hal-01174251</identifier> <identifier>https://hal-univ-perp.archives-ouvertes.fr/hal-01174251/document</identifier> <identifier>https://hal-univ-perp.archives-ouvertes.fr/hal-01174251/file/peerj-1034.pdf</identifier> <source>https://hal-univ-perp.archives-ouvertes.fr/hal-01174251</source> <source>PeerJ, PeerJ, 2015, 〈10.7717/peerj.1034〉</source> <identifier>DOI : 10.7717/peerj.1034</identifier> <relation>info:eu-repo/semantics/altIdentifier/doi/10.7717/peerj.1034</relation> <language>en</language> <subject lang=en>Crustose coralline algae</subject> <subject lang=en>Disease</subject> <subject lang=en>Cell death</subject> <subject lang=en>Boring fauna</subject> <subject lang=en>Lesion</subject> <subject lang=en>Histopathology</subject> <subject lang=en>Regeneration</subject> <subject>[SDE.BE] Environmental Sciences/Biodiversity and Ecology</subject> <type>info:eu-repo/semantics/article</type> <type>Journal articles</type> <description lang=en>Crustose coralline algae (CCA) are major benthic calcifiers that play crucial roles in marine ecosystems, particularly coral reefs. Over the past two decades, epizootics have been reported for several CCA species on coral reefs worldwide. However, their causes remain often unknown in part because few studies have investigated CCA pathologies at a microscopic scale. We studied the cellular changes associated with two syndromes: Coralline White Band Syndrome (CWBS) and Coralline White Patch Disease (CWPD) from samples collected in Curaçao , southern Caribbean. Healthy-looking tissue of diseased CCA did not differ from healthy tissue of healthy CCA. In diseased tissues of both pathologies, the three characteristic cell layers of CCA revealed cells completely depleted of protoplasmic content, but presenting an intact cell wall. In addition, CWBS showed a transition area between healthy and diseased tissues consisting of cells partially deprived of protoplasmic material, most likely corresponding to the white band characterizing the disease at the macroscopic level. This transition area was absent in CWPD. Regrowth at the lesion boundary were sometimes observed in both syndromes. Tissues of both healthy and diseased CCA were colonised by diverse boring organisms. Fungal infections associated with the diseased cells were not seen. However, other bioeroders were more abundant in diseased vs healthy CCA and in diseased vs healthy-looking tissues of diseased CCA. Although their role in the pathogenesis is unclear, this suggests that disease increases CCA susceptibility to bioerosion. Further investigations using an integrated approach are needed to carry out the complete diagnosis of these diseases.</description> <rights>http://creativecommons.org/licenses/by/</rights> <date>2015</date> </dc> </metadata> </record> </GetRecord> </OAI-PMH>