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<datestamp>2017-12-21</datestamp>
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<publisher>HAL CCSD</publisher>
<title lang=en>Is Exercise-Induced Arterial Hypoxemia in Triathletes Dependent on Exercise Modality ?</title>
<creator>Galy, Olivier</creator>
<creator>Boussana, Alain</creator>
<creator>Hue, Olivier</creator>
<creator>Le Gallais, Daniel</creator>
<creator>Prefaut, Christian</creator>
<contributor>Adaptations au Climat Tropical, Exercice et Santé (ACTES) ; Université des Antilles et de la Guyane (UAG)</contributor>
<contributor>Laboratoire de Physiologie des Interactions ; CHU Arnaud de Villeneuve</contributor>
<contributor>Centre d'Optimisation de la Performance Motrice ; Université Montpellier 1 (UM1)</contributor>
<contributor>Euromov (EuroMov) ; Centre Hospitalier Régional Universitaire [Montpellier] (CHRU Montpellier) - Institut National de la Santé et de la Recherche Médicale (INSERM) - Université de Montpellier (UM)</contributor>
<contributor>Interface Biopsychosociale des A.P.A. ; Université Montpellier 1 (UM1)</contributor>
<description>International audience</description>
<source>Internation Journal of Sports Medicine</source>
<identifier>hal-00720806</identifier>
<identifier>https://hal.univ-antilles.fr/hal-00720806</identifier>
<identifier>https://hal.univ-antilles.fr/hal-00720806/document</identifier>
<identifier>https://hal.univ-antilles.fr/hal-00720806/file/sm290.pdf</identifier>
<source>https://hal.univ-antilles.fr/hal-00720806</source>
<source>Internation Journal of Sports Medicine, 2005, 26 (9), pp.719-726</source>
<language>en</language>
<subject lang=en>Pulmonary gas exchanges</subject>
<subject lang=en>EIAH</subject>
<subject lang=en>DLCO</subject>
<subject lang=en>endurance training</subject>
<subject lang=en>beathing pattern</subject>
<subject lang=en>triathlon</subject>
<subject>[SDV.OT] Life Sciences [q-bio]/Other [q-bio.OT]</subject>
<type>info:eu-repo/semantics/article</type>
<type>Journal articles</type>
<description lang=en>To determine whether exercise modality affects arterial hypoxemia (EIAH) during training-intensity exercise, 13 triathletes performed 20 min of cycling (C) followed by 20 min of running (R): C-R, and two weeks later, 20 min of R followed by 20 min of C:R-C. Each trial was performed at an intensity slightly above the ventilatory threshold and close to the daily training intensity (75 % of VO2max). Ventilatory data were collected continuously using an automated breath-by-breath system. Partial pressure of oxygen in arterial blood (PaO2) was measured after each C and R segment and arterial oxyhemoglobin saturation (SpO2) was monitored continuously via pulse oximetry. The metabolic rate was similar across modalities and trials, i.e., C-R (53.8 +/- 3.8 vs. 51.1 +/- 5.3 ml.min(-1).kg(-1)) and R-C (52.2 +/- 4.5 vs. 53.2 +/- 4.6 ml.min(-1).kg (-1)). EIAH showed significantly greater severity for R compared to C irrespective of the order (p < 0.05 for both trials). R values of PaO2 (and SpO2) for C-R and R-C were 88.7 +/- 6.0 mm Hg (93.0 +/- 0.6 % SpO2) and 86.6 +/- 7.3 mm Hg (93.5 +/- 0.6 % SpO2) and C values were 93.7 +/- 8.4 mm Hg (95.4 +/- 0.4 % SpO2) and 91.4 +/- 5.4 mm Hg (94.8 +/- 0.3 % SpO2). R ventilatory data described a significantly different breathing pattern than C, with higher respiratory rate (35.9 b.min(-1) vs. 51.1 b.min(-1) for C-R, p < 0.01; and 50.0 b.min(-1) vs. 41.5 b.min(-1) for R-C, p < 0.01) and lower tidal volume (2636 ml vs. 2282 ml for C-R, p < 0.02 and 2272 ml vs. 2472 ml for R-C, p < 0.05). We concluded that EIAH was greater during running than cycling for a similar metabolic rate corresponding to training intensity and that EIAH could thus be considered dependent on exercise modality.</description>
<date>2005-11</date>
</dc>
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